Preliminary studies suggest conolidine may well inhibit unique ion channels, decreasing neuronal excitability and restricting ache indicators. This mechanism is especially related in neuropathic ache, in which irregular signaling triggers persistent discomfort. Moreover, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to ache perception. Computational do... https://collinsg394ari9.wikirecognition.com/user